Also indexed as: Para-aminobenzoic Acid
What does it do? PABA is the abbreviation for
para-aminobenzoic acid, a compound that is an essential nutrient for microorganisms and some
animals, but has not been shown to be essential for people. PABA is considered by some to be a
member of the vitamin B-complex, though its
actions differ widely from other B vitamins. PABA has been reported to enhance the effects of
cortisone.1 It may also prevent or even reverse accumulation of abnormal fibrous
tissue.
The most well-known property of PABA is as an effective sunscreen, when used topically.
Oral PABA supplementation has not been shown to possess any sunscreening
properties.2
An isolated trial published in 1942 reported that 12 of 16 infertile women were able to become pregnant after supplementing with 100 mg of PABA taken four
times per day for three to seven months.3 The effect of PABA on fertility has not
been studied in modern research.
Researchers have attempted to discover whether large amounts of PABA would be helpful in
various connective tissue disorders. Although preliminary studies have reported that PABA (12
grams per day) was helpful to people with scleroderma,4 5 6 a
double-blind trial found that supplementation with PABA did not lead to
improvement.7
Older published reports of uncontrolled investigations suggest that PABA may be helpful in
a variety of conditions, including dermatomyositis,8 Peyronie’s disease
(accumulation of abnormal fibrous tissue in the penis),9 pemphigus (a severe
blistering disease),10 and vitiligo (a
disorder in which patches of skin lose their pigmentation).11 However, PABA was
reported to cause vitiligo in one report.12
Older preliminary reports found that PABA darkened gray hair in a minority of elderly (but
not younger) people.13 In these trials between 200 and 600 mg of PABA was taken per
day for several months, in some cases accompanied by other B vitamins. However, at least one other study found that
PABA did not darken gray hair.14 Therefore, the evidence supporting the use of PABA
as a way to return gray hair to its original color remains very weak.
Where is it found? PABA is found in grains and foods of animal origin.
PABA has been used in
connection with the following conditions (refer to the individual
health concern for complete information):
Who is likely to be deficient? Deficiencies of PABA have not
been described in humans, and most nutritionists do not consider it an essential nutrient.
How much is usually taken? Small amounts of PABA are present
in some B-complex vitamins and multivitamin formulas. The amount of PABA used in the studies
described above ranged from 300 mg to 12 grams per day. Anyone taking more than 400 mg of PABA
per day should consult a physician.
Are there any side effects or interactions? No serious side
effects have been reported with 300–400 mg per day. Larger amounts (such as 8 grams per
day or more) may cause low blood sugar, rash, fever,
and (on rare occasions) liver damage.15 One report exists of vitiligo appearing after ingestion of large amounts of
PABA16 and use of amounts over 20 grams per day in small children has resulted in
deaths.17 There is also a report of a death from toxic hepatitis in a person with
lupus, who took as much as 48 grams per day for six days, followed by 8 grams per day for
seven months.18
No interactions between PABA and other nutrients have been reported. However, PABA
interferes with sulfa drugs (a class of antibiotics) and
therefore should not be taken when these medications are being used.
Are there any drug interactions? Certain medications may
interact with PABA. Refer to the drug interactions safety
check for a list of those medications.
References:
1. Wiesel LL, Barritt AS, Stumpe WM. The synergistic action of
para-aminobenzoic acid and cortisone in the treatment of rheumatoid arthritis. Am J Med
Sci 1951;222:243–8.
2. Willis I, Kligman AM. Aminobenzoic acid and its esters. The quest for
more effective sunscreens. Arch Dermatol 1970;102:405–17.
3. Sieve BF. The clinical effects of a new B-complex factor,
para-aminobenzoic acid, on pigmentation and fertility. South Med Surg
1942(March);104:135–9.
4. Zarafonetis CJD. The treatment of scleroderma: results of potassium
para-aminobenzoate therapy in 104 cases. In: Mills LC, Moyer JH eds. Inflammation and
Diseases of Connective Tissue. Philadelphia: W. B. Saunders Co. 1961, 688–96.
5. Zarafonetis CJD, Dabich L, Negri D, et al. Retrospective studies in
scleroderma: effect of potassium para-aminobenzoate on survival. J Clin Epidemiol
1988;41:193–205.
6. Zarafonetis CJ, Dabich L, Devol EB, et al. Retrospective studies in
scleroderma: pulmonary findings and effect of potassium p-aminobenzoate on vital capacity.
Respiration 1989;56:22–33.
7. Clegg DO, Reading JC, Mayes MD, et al. Comparison of aminobenzoate
potassium and placebo in the treatment of scleroderma. J Rheumatol
1994;21:105–10.
8. Grace WJ, Kennedy RJ, Formato A. Therapy of scleroderma and
dermatomyositis. NY State J Med 1963;63:140–4.
9. Zarafonetis CJD. Treatment of Peyronie’s disease with potassium
para-aminobenzoate. J Urol 1959;81:770–2.
10. Zarafonetis CJD, Curtis AC, Shaw JM. Treatment of pemphigus with
potassium para-aminobenzoate. Am J Med Sci 1956;231:30–50.
11. Sieve BF. Further investigations in the treatment of vitiligo.
Virginia Med Monthly 1945(January):6–17.
12. Hughes CG. Oral PABA and vitiligo. J Am Acad Dermatol
1983;9:770 [letter].
13. Gaby AR. The story of PABA. Nutr Healing
1997;March:3–4, 11 [review].
14. Zarafonetis CJD. Darkening of gray hair during para-amino-benzoic
acid therapy. J Invest Dermatol 1950;15:399–401.
15. Kantor GR, Ratz JL. Liver toxicity from potassium para-aminobenzoate.
J Am Acad Dermatol 1985;13:671–2.
16. Hughes CG. Oral PABA and vitiligo. J Am Acad Dermatol
1983;9:770 [letter].
17. Worobec S, LaChine A. Dangers of orally administered
para-aminobenzoic acid. JAMA 1984;251:2348.
18. Zarafonetis CJD, Grekin RH, Curtis AC, et al. Further studies on the
treatment of lupus erythematosus with sodium para-aminobenzoate. J Invest Dermatol
1948;11:359.
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The information presented in Healthnotes is for informational
purposes only. It is based on scientific studies (human, animal, or in vitro),
clinical experience, or traditional usage as cited in each article. The results reported may
not necessarily occur in all individuals. For many of the conditions discussed, treatment with
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before making any changes in prescribed medications. Information expires December 2003.
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